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Theyinduce prolonged partial depolarization of theregion around muscle end plate Na+ channelsget inactivated (because transmembrane potentialdrops to about –50 mV) ACh released frommotor nerve endings is unable to generatepropagated MAP flaccid paralysis inmammals. (2002) Molecularsignatures of brain injury after intracerebral hemorrhage. acetaminophen pharmacokinet-ics in neonates after multiple doses.

Theaxon leaves the cell body at the axon hillock.

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